A Phase I/II Study Evaluating the Safety and the Efficacy of SMART101 Injection to Accelerate Immune Reconstitution After T Cell Depleted Allogeneic Hematopoietic Stem Cell Transplantation in Pediatric and Adult Patients With Hematological Malignancies

Who is this study for? Adult and pediatric patients with relapsed/refractory acute leukemia
What treatments are being studied? Allogeneic T cell progenitors, cultured ex-vivo
Status: Recruiting
Location: See location...
Intervention Type: Biological
Study Type: Interventional
Study Phase: Phase 1/Phase 2
SUMMARY

The purpose of this study is to evaluate the safety and the efficacy of SMART101 (Human T Lymphoid Progenitor (HTLP)) injection to accelerate immune reconstitution after T cell depleted allogeneic hematopoietic stem cell transplantation (HSCT) in adult and pediatric patients with hematological malignancies.

Eligibility
Participation Requirements
Sex: All
Healthy Volunteers: f
View:

∙ Group A (adults):

• Adult patients affected by:

‣ Acute leukemia (AML, ALL) defined as:

• Acute Myeloid Leukemia (AML):

‣ High risk AML in CR1; any adverse genetic abnormality, secondary or therapy related AML excluding good risk genetic abnormalities

⁃ Chemo-refractory relapse (MRD+)

⁃ ≥ CR2

∙ Acute Lymphoblastic Leukemia (ALL):

‣ Chemo-refractory relapse (MRD+)

⁃ High risk ALL in CR1; Philadelphia (like) or any poor risk feature

⁃ ≥ CR2

∙ Acute leukemia of ambiguous lineage:

‣ ≥ CR1 with a minimal residual disease (MRD) \<5% (flow cytometry, molecular and/or cytogenetics accepted)

⁃ Myelodysplastic Syndrome (MDS) with least one of the following:

• Revised International Prognostic Scoring System risk score of intermediate or higher at the time of transplant evaluation.

∙ Life-threatening cytopenia.

∙ Karyotype or genomic changes that indicate high risk for progression to acute myelogenous leukemia, including abnormalities of chromosome 7 or 3, mutations of TP53, or complex or monosomal karyotype.

∙ Therapy related disease or disease evolving from other malignant processes.

• Patient eligible for a T-depleted allogeneic HSCT

• Age ≥ 18y and clinical condition compatible with allogeneic stem cell transplantation

• Karnofsky index ≥ 70% prior to conditioning regimen

• Patients with normal organ function prior to conditioning regimen

∙ Group B (pediatrics):

• Pediatric patients affected by acute leukemia defined as:

‣ Acute Myeloid Leukemia (AML):

• High risk AML in CR1; any adverse genetic abnormality, secondary or therapy related AML excluding good risk genetic abnormalities,

∙ Chemo-refractory relapse (MRD+)

∙ ≥ CR2

⁃ Acute Lymphoblastic Leukemia (ALL):

• Chemo-refractory relapse (MRD+)

∙ High risk ALL in CR1; Philadelphia (like) or any poor risk feature

∙ ≥ CR2

⁃ Acute leukemia of ambiguous lineage:

• ≥ CR1 with a minimal residual disease (MRD) \<5% (flow cytometry, molecular and/or cytogenetics accepted)

• Patient eligible for a T-depleted allogeneic HSCT

• Age \< 18y at the time of inclusion

• Absence of a matched sibling donor (MSD)

• Lansky ≥ 70% / Karnofsky performance status ≥ 70% prior to conditioning regimen

• Patients with normal organ function prior to conditioning regimen

Locations
United States
New York
Memorial Sloan Kettering Cancer Center (MSKCC)
RECRUITING
New York
Contact Information
Primary
Frédéric LEHMANN, MD
frederic.lehmann@smart-immune.com
+32 (0) 492 46 23 55
Backup
Laura SIMONS, MD, PhD
laura.simons@smart-immune.com
Time Frame
Start Date: 2022-03-31
Estimated Completion Date: 2027-05
Participants
Target number of participants: 36
Treatments
Experimental: Adult patients affected by hematological malignancies
Adult patients affected by acute leukemia (AML, ALL or acute leukemia of ambiguous lineage) or myelodysplastic syndrome eligible for a T depleted allogeneic HSCT
Experimental: Pediatric patients affected by hematological malignancies
Pediatric patients affected by acute leukemia (AML, ALL or acute leukemia of ambiguous lineage) eligible for a T depleted allogeneic HSCT
Related Therapeutic Areas
Sponsors
Leads: Smart Immune SAS

This content was sourced from clinicaltrials.gov